Gilles Dubuis

Publications | Mémoires et thèses

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14 publications

2022 | 2021 | 2020 | 2019 | 2014 | 2013 | 2012 | 2011 | 2009 | 2007 | 2005 |
HDLs extract lipophilic drugs from cells.
Zheng A., Dubuis G., Georgieva M., Mendes Ferreira C.S., Serulla M., Del Carmen Conde Rubio M., Trofimenko E., Mercier T., Decosterd L., Widmann C., 2022/03/01. Journal of cell science, 135 (5) pp. jcs258644. Peer-reviewed.
APOBEC3C, a nucleolar protein induced by genotoxins, is excluded from DNA damage sites.
Constantin D., Dubuis G., Conde-Rubio MDC, Widmann C., 2022/02. The FEBS journal, 289 (3) pp. 808-831. Peer-reviewed.
Genetic, cellular, and structural characterization of the membrane potential-dependent cell-penetrating peptide translocation pore.
Trofimenko E., Grasso G., Heulot M., Chevalier N., Deriu M.A., Dubuis G., Arribat Y., Serulla M., Michel S., Vantomme G. et al., 2021/10/29. eLife, 10 pp. e69832. Peer-reviewed.
Loss-of-function of the long non-coding RNA A830019P07Rik in mice does not affect insulin expression and secretion.
Guay C., Abdulkarim B., Tan J.Y., Dubuis G., Rütti S., Ross Laybutt D., Widmann C., Regazzi R., Marques A.C., 2020/04/14. Scientific reports, 10 (1) p. 6413. Peer-reviewed.
CRISPR/Cas9 genome-wide screening identifies KEAP1 as a sorafenib, lenvatinib, and regorafenib sensitivity gene in hepatocellular carcinoma.
Zheng A., Chevalier N., Calderoni M., Dubuis G., Dormond O., Ziros P.G., Sykiotis G.P., Widmann C., 2019/12/17. Oncotarget, 10 (66) pp. 7058-7070. Peer-reviewed.
 
The PI3K/Akt pathway is not a main driver in HDL-mediated cell protection.
Zheng A., Dubuis G., Ferreira CSM, Pétremand J., Vanli G., Widmann C., 2019/10. Cellular signalling, 62 p. 109347. Peer-reviewed.
 
The activity of the anti-apoptotic fragment generated by the caspase-3/p120 RasGAP stress-sensing module displays strict Akt isoform specificity.
Vanli G., Peltzer N., Dubuis G., Widmann C., 2014. Cellular Signalling, 26 (12) pp. 2992-2997. Peer-reviewed.
HDLs protect the MIN6 insulinoma cell line against tunicamycin-induced apoptosis without inhibiting ER stress and without restoring ER functionality.
Puyal J., Pétremand J., Dubuis G., Rummel C., Widmann C., 2013. Molecular and Cellular Endocrinology, 381 (1-2) pp. 291-301. Peer-reviewed.
Caspase-3 Protects Stressed Organs against Cell Death.
Khalil H., Peltzer N., Walicki J., Yang J.Y., Dubuis G., Gardiol N., Held W., Bigliardi P., Marsland B., Liaudet L. et al., 2012. Molecular and Cellular Biology, 32 (22) pp. 4523-4533. Peer-reviewed.
RasGAP-derived fragment N increases the resistance of beta cells towards apoptosis in NOD mice and delays the progression from mild to overt diabetes.
Bulat N., Jaccard E., Peltzer N., Khalil H., Yang J.Y., Dubuis G., Widmann C., 2011. PLoS One, 6 (7) pp. e22609.
Expression of the NH(2)-terminal fragment of RasGAP in pancreatic beta-cells increases their resistance to stresses and protects mice from diabetes.
Yang J.Y., Walicki J., Jaccard E., Dubuis G., Bulat N., Hornung J.P., Thorens B., Widmann C., 2009. Diabetes, 58 (11) pp. 2596-2606. Peer-reviewed.
 
Role of the sub-cellular localization of RasGAP fragment N2 for its ability to sensitize cancer cells to genotoxin-induced apoptosis.
Annibaldi A., Michod D., Vanetta L., Cruchet S., Nicod P., Dubuis G., Bonvin C., Widmann C., 2009. Experimental Cell Research, 315 (12) pp. 2081-2091. Peer-reviewed.
 
Splice variant-specific stabilization of JNKs by IB1/JIP1.
Yang J.Y., Moulin N., van Bemmelen M.X., Dubuis G., Tawadros T., Haefliger J.A., Waeber G., Widmann C., 2007. Cellular signalling, 19 (10) pp. 2201-7. Peer-reviewed.
 
Impaired Akt activity down-modulation, caspase-3 activation, and apoptosis in cells expressing a caspase-resistant mutant of RasGAP at position 157
Yang J. Y., Walicki J., Michod D., Dubuis G., Widmann C., 2005/08. Molecular Biology of the Cell, 16 (8) pp. 3511-20.
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