Curdin Conrad

Publications | Mémoires et thèses

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51 publications

2019 | 2018 | 2017 | 2016 | 2015 | 2014 | 2013 | 2012 | 2011 | 2010 | 2009 | 2008 | 2007 | 2006 | 2005 | 2004 |
 
IL-17E (IL-25) Enhances Innate Immune Responses during Skin Inflammation.
Senra L., Mylonas A., Kavanagh R.D., Fallon P.G., Conrad C., Borowczyk-Michalowska J., Wrobel L.J., Kaya G., Yawalkar N., Boehncke W.H. et al., 2019/08. The Journal of investigative dermatology, 139 (8) pp. 1732-1742.e17. Peer-reviewed.
 
Secukinumab drug survival in patients with psoriasis: A multicenter, real-world, retrospective study.
Torres T., Balato A., Conrad C., Conti A., Dapavo P., Ferreira P., Gaiani F., Leite L., Malagoli P., Mendes-Bastos P. et al., 2019/07. Journal of the American Academy of Dermatology, 81 (1) pp. 273-275. Peer-reviewed.
 
Gender and age significantly determine patient needs and treatment goals in psoriasis - a lesson for practice.
Maul J.T., Navarini A.A., Sommer R., Anzengruber F., Sorbe C., Mrowietz U., Drach M., Blome C., Boehncke W.H., Thaci D. et al., 2019/04. Journal of the European Academy of Dermatology and Venereology, 33 (4) pp. 700-708. Peer-reviewed.
 
Psoriasis paradoxal induit par anti-TNF – un challenge en clinique [Paradoxical psoriasis induced by anti-TNF - a clinical challenge]
Nidegger A., Mylonas A., Conrad C., 2019/03/27. Revue medicale suisse, 15 (644) pp. 668-671. Peer-reviewed.
 
Human "T<sub>H</sub>9" cells are a subpopulation of PPAR-γ<sup>+</sup> T<sub>H</sub>2 cells.
Micossé C., von Meyenn L., Steck O., Kipfer E., Adam C., Simillion C., Seyed Jafari S.M., Olah P., Yawlkar N., Simon D. et al., 2019/01/18. Science immunology, 4 (31). Peer-reviewed.
Regulatory T Cells Restrain Pathogenic T Helper Cells during Skin Inflammation.
Hartwig T., Zwicky P., Schreiner B., Yawalkar N., Cheng P., Navarini A., Dummer R., Flatz L., Conrad C., Schlapbach C. et al., 2018/12/26. Cell reports, 25 (13) pp. 3564-3572.e4. Peer-reviewed.
 
Psoriasis: from Pathogenesis to Targeted Therapies.
Conrad C., Gilliet M., 2018/02. Clinical reviews in allergy & immunology, 54 (1) pp. 102-113. Peer-reviewed.
 
TNF blockade induces a dysregulated type I interferon response without autoimmunity in paradoxical psoriasis.
Conrad C., Di Domizio J., Mylonas A., Belkhodja C., Demaria O., Navarini A.A., Lapointe A.K., French L.E., Vernez M., Gilliet M., 2018/01/02. Nature communications, 9 (1) p. 25. Peer-reviewed.
 
Diversification of human plasmacytoid predendritic cells in response to a single stimulus.
Alculumbre S.G., Saint-André V., Di Domizio J., Vargas P., Sirven P., Bost P., Maurin M., Maiuri P., Wery M., Roman M.S. et al., 2018/01. Nature immunology, 19 (1) pp. 63-75. Peer-reviewed.
Psoriasis: Classical vs. Paradoxical. The Yin-Yang of TNF and Type I Interferon.
Mylonas A., Conrad C., 2018. Frontiers in immunology, 9 p. 2746. Peer-reviewed.
 
Swiss S1 guideline for the treatment of rosacea.
Anzengruber F., Czernielewski J., Conrad C., Feldmeyer L., Yawalkar N., Häusermann P., Cozzio A., Mainetti C., Goldblum D., Läuchli S. et al., 2017/11. Journal of the European Academy of Dermatology and Venereology, 31 (11) pp. 1775-1791. Peer-reviewed.
 
Mise au point sur la rectocolite ulcéro-hémorragique en 2017 [Management of ulcerative colitis in 2017]
Moschouri E., Hessler R., Pittet V., Velin D., Conrad C., Hahnloser D., Gié O., Michetti P., Schoepfer A.M., Maillard M.H., 2017/08/30. Revue medicale suisse, 13 (572) pp. 1480-1486. Peer-reviewed.
Interleukin 23-Helper T Cell 17 Axis as a Treatment Target for Pityriasis Rubra Pilaris.
Feldmeyer L., Mylonas A., Demaria O., Mennella A., Yawalkar N., Laffitte E., Hohl D., Gilliet M., Conrad C., 2017/04/01. JAMA dermatology, 153 (4) pp. 304-308. Peer-reviewed.
 
Xenotransplantation Model of Psoriasis.
Di Domizio J., Conrad C., Gilliet M., 2017. Methods in molecular biology, 1559 pp. 83-90. Peer-reviewed.
Targeting CD8(+) T cells prevents psoriasis development.
Di Meglio P., Villanova F., Navarini A.A., Mylonas A., Tosi I., Nestle F.O., Conrad C., 2016/07. The Journal of allergy and clinical immunology, 138 (1) pp. 274-276.e6. Peer-reviewed.
 
Efficacy and Survival of Systemic Psoriasis Treatments: An Analysis of the Swiss Registry SDNTT.
Maul J.T., Djamei V., Kolios AGA, Meier B., Czernielewski J., Jungo P., Yawalkar N., Mainetti C., Laffitte E., Spehr C. et al., 2016. Dermatology (Basel, Switzerland), 232 (6) pp. 640-647. Peer-reviewed.
 
PASS Syndrome: An IL-1-Driven Autoinflammatory Disease.
Leuenberger M., Berner J., Di Lucca J., Fischer L., Kaparos N., Conrad C., Hohl D., So A., Gilliet M., 2016. Dermatology (Basel, Switzerland), 232 (2) pp. 254-258. Peer-reviewed.
 
Superiority in Quality of Life Improvement of Biologics over Conventional Systemic Drugs in a Swiss Real-Life Psoriasis Registry.
Jungo P., Maul J.T., Djamei V., von Felten S., Kolios AGA, Czernielewsk J., Yawalkar N., Odermatt O., Laffitte E., Anliker M. et al., 2016. Dermatology (Basel, Switzerland), 232 (6) pp. 655-663. Peer-reviewed.
 
Swiss S1 Guidelines on the Systemic Treatment of Psoriasis Vulgaris.
Kolios A.G., Yawalkar N., Anliker M., Boehncke W.H., Borradori L., Conrad C., Gilliet M., Häusermann P., Itin P., Laffitte E. et al., 2016. Dermatology (basel, Switzerland), 232 (4) pp. 385-406. Peer-reviewed.
 
Cationic antimicrobial peptides in psoriatic skin cooperate to break innate tolerance to self-DNA.
Lande R., Chamilos G., Ganguly D., Demaria O., Frasca L., Durr S., Conrad C., Schröder J., Gilliet M., 2015. European Journal of Immunology, 45 (1) pp. 203-213. Peer-reviewed.
 
Corrigendum: the antimicrobial peptide LL37 is a T-cell autoantigen in psoriasis.
Lande R., Botti E., Jandus C., Dojcinovic D., Fanelli G., Conrad C., Chamilos G., Feldmeyer L., Marinari B., Chon S. et al., 2015..
TH17 cells promote microbial killing and innate immune sensing of DNA via interleukin 26.
Meller S., Di Domizio J., Voo K.S., Friedrich H.C., Chamilos G., Ganguly D., Conrad C., Gregorio J., Le Roy D., Roger T. et al., 2015. Nature Immunology, 16 (9) pp. 970-979. Peer-reviewed.
 
The dermatologists' role in managing psoriatic arthritis: results of a swiss delphi exercise intended to improve collaboration with rheumatologists.
Boehncke W.H., Anliker M.D., Conrad C., Dudler J., Hasler F., Hasler P., Häusermann P., Kyburz D., Laffitte E., Michel B.A. et al., 2015. Dermatology, 230 (1) pp. 75-81. Peer-reviewed.
Purpura of the face and neck: an atypical clinical presentation revealing a hepatosplenic T cell lymphoma
Kuonen F., Bucher M., de Leval L., Vernez M., Gilliet M., Conrad C., Feldmeyer L., 2014. Case Reports in Dermatology, 6 (1) pp. 37-42. Peer-reviewed.
 
The antimicrobial peptide LL37 is a T-cell autoantigen in psoriasis.
Lande R., Botti E., Jandus C., Dojcinovic D., Fanelli G., Conrad C., Chamilos G., Feldmeyer L., Marinari B., Chon S. et al., 2014. Nature Communications, 5 p. 5621. Peer-reviewed.
Plasmacytoid dendritic cells and regulatory T cells in the tumor microenvironment: A dangerous liaison.
Conrad C., Gilliet M., 2013/05. OncoImmunology, 2 (5) pp. e23887.
 
Anti-TNF Therapy in the Treatment of Psoriasis in a Patient with Acute-on-Chronic Pancreatitis.
Clayton H., Flatz L., Vollenweider-Roten S., Schoepfer A., Gilliet M., Conrad C., 2013. Dermatology, 227 (3) pp. 193-196.
Role of T-cell-mediated inflammation in psoriasis: pathogenesis and targeted therapy
Flatz L., Conrad C., 2013. Psoriasis: Targets and Therapy, 3 pp. 1-10.
 
Zur Krankheitslast von Psoriasis-Patienten in der Schweiz
Kolios A.G.A., Czernielewski J., Häusermann P., Yawalkar N., Navarini A.A., Thurneysen S., Laffitte E., Djamei V., Spehr C., Anliker M. et al., 2013. Dermatologica Helvetica, 25 (10) pp. 28-31.
 
Plasmacytoid dendritic cells promote immunosuppression in ovarian cancer via ICOS costimulation of Foxp3(+) T-regulatory cells.
Conrad C., Gregorio J., Wang Y.H., Ito T., Meller S., Hanabuchi S., Anderson S., Atkinson N., Ramirez P.T., Liu Y.J. et al., 2012. Cancer Research, 72 (20) pp. 5240-5249.
 
Type I IFNs at the interface between cutaneous immunity and epidermal remodeling.
Conrad C., Gilliet M., 2012. Journal of Investigative Dermatology, 132 (7) pp. 1759-1762.
 
Neutrophils activate plasmacytoid dendritic cells by releasing self-DNA-peptide complexes in systemic lupus erythematosus.
Lande R., Ganguly D., Facchinetti V., Frasca L., Conrad C., Gregorio J., Meller S., Chamilos G., Sebasigari R., Riccieri V. et al., 2011. Science Translational Medicine, 3 (73) pp. 73ra19.
Estimation of cost-of-illness in patients with psoriasis in Switzerland.
Navarini A.A., Laffitte E., Conrad C., Piffaretti P., Brock E., Ruckdaeschel S., Trüeb R.M., 2010/02/06. Swiss medical weekly, 140 (5-6) pp. 85-91. Peer-reviewed.
 
Cutting edge: A critical functional role for IL-23 in psoriasis.
Tonel G., Conrad C., Laggner U., Di Meglio P., Grys K., McClanahan T.K., Blumenschein W.M., Qin J.Z., Xin H., Oldham E. et al., 2010. Journal of Immunology, 185 (10) pp. 5688-5691.
 
Plasmacytoid dendritic cells sense skin injury and promote wound healing through type I interferons.
Gregorio J., Meller S., Conrad C., Di Nardo A., Homey B., Lauerma A., Arai N., Gallo R.L., Digiovanni J., Gilliet M., 2010. Journal of Experimental Medicine, 207 (13) pp. 2921-2930.
 
Trauma as triggering factor for development of melanocytic nevi.
Navarini A.A., Kolm I., Calvo X., Kamarashev J., Kerl K., Conrad C., French L.E., Braun R.P., 2010. Dermatology, 220 (4) pp. 291-296.
 
Interplay between keratinocytes and immune cells--recent insights into psoriasis pathogenesis.
Tonel G., Conrad C., 2009. International Journal of Biochemistry and Cell Biology, 41 (5) pp. 963-968.
 
Melanoma and innate immunity--aActive inflammation or just erroneous attraction? Melanoma as the source of leukocyte-attracting chemokines.
Navarini-Meury A.A., Conrad C., 2009. Seminars in Cancer Biology, 19 (2) pp. 84-91.
 
Plasmacytoid dendritic cells in the skin: to sense or not to sense nucleic acids.
Conrad C., Meller S., Gilliet M., 2009. Seminars in Immunology, 21 (3) pp. 101-109.
 
Skin problems associated with pegylated liposomal doxorubicin-more than palmoplantar erythrodysesthesia syndrome.
Mangana J., Zipser M.C., Conrad C., Oberholzer P.A., Cozzio A., Knuth A., French L.E., Dummer R., 2008. European Journal of Dermatology, 18 (5) pp. 566-570.
 
Alpha1beta1 integrin is crucial for accumulation of epidermal T cells and the development of psoriasis.
Conrad C., Boyman O., Tonel G., Tun-Kyi A., Laggner U., de Fougerolles A., Kotelianski V., Gardner H., Nestle F.O., 2007. Nature medicine, 13 (7) pp. 836-842. Peer-reviewed.
 
The pathogenic role of tissue-resident immune cells in psoriasis.
Boyman O., Conrad C., Tonel G., Gilliet M., Nestle F.O., 2007. Trends in Immunology, 28 (2) pp. 51-57. Peer-reviewed.
 
Animal models of psoriasis and psoriatic arthritis: an update.
Conrad C., Nestle F.O., 2006. Current Rheumatology Reports, 8 (5) pp. 342-347.
 
Toll-like receptor engagement converts T-cell autoreactivity into overt autoimmune disease
Lang K. S., Recher M., Junt T., Navarini A. A., Harris N. L., Freigang S., Odermatt B., Conrad C., Ittner L. M., Bauer S. et al., 2005/02. Nature Medicine, 11 (2) pp. 138-45.
 
CD4+CD56+ hematodermic neoplasms bear a plasmacytoid dendritic cell phenotype.
Urosevic M., Conrad C., Kamarashev J., Asagoe K., Cozzio A., Burg G., Dummer R., 2005. Human Pathology, 36 (9) pp. 1020-1024.
 
Dendritic-cell-based therapeutic vaccination against cancer.
Nestle F.O., Farkas A., Conrad C., 2005. Current Opinion in Immunology, 17 (2) pp. 163-169.
 
Disease-independent skin recruitment and activation of plasmacytoid predendritic cells following imiquimod treatment.
Urosevic M., Dummer R., Conrad C., Beyeler M., Laine E., Burg G., Gilliet M., 2005. Journal of the National Cancer Institute, 97 (15) pp. 1143-1153.
 
Plasmacytoid predendritic cells initiate psoriasis through interferon-alpha production.
Nestle F.O., Conrad C., Tun-Kyi A., Homey B., Gombert M., Boyman O., Burg G., Liu Y.J., Gilliet M., 2005. Journal of Experimental Medicine, 202 (1) pp. 135-143.
 
Pyoderma gangrenosum.
Conrad C., Trüeb R.M., 2005. Journal der Deutschen Dermatologischen Gesellschaft = Journal of the German Society of Dermatology, 3 (5) pp. 334-342.
 
Psoriasis triggered by toll-like receptor 7 agonist imiquimod in the presence of dermal plasmacytoid dendritic cell precursors.
Gilliet M., Conrad C., Geiges M., Cozzio A., Thürlimann W., Burg G., Nestle F.O., Dummer R., 2004. Archives of Dermatology, 140 (12) pp. 1490-1495.
 
Spontaneous development of psoriasis in a new animal model shows an essential role for resident T cells and tumor necrosis factor-alpha.
Boyman O., Hefti H.P., Conrad C., Nickoloff B.J., Suter M., Nestle F.O., 2004. Journal of Experimental Medicine, 199 (5) pp. 731-736.
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