Jean Halbert

Publications | Mémoires et thèses

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12 publications

2020 | 2018 | 2017 | 2014 | 2013 | 2011 | 2010 | 2007 | 2005 |
 
Expression Evolution of Ancestral XY Gametologs across All Major Groups of Placental Mammals.
Martínez-Pacheco M., Tenorio M., Almonte L., Fajardo V., Godínez A., Fernández D., Cornejo-Páramo P., Díaz-Barba K., Halbert J., Liechti A. et al., 2020/11/03. Genome biology and evolution, 12 (11) pp. 2015-2028. Peer-reviewed.
Repurposing of promoters and enhancers during mammalian evolution.
Carelli F.N., Liechti A., Halbert J., Warnefors M., Kaessmann H., 2018/10/04. Nature communications, 9 (1) p. 4066. Peer-reviewed.
Convergent origination of a Drosophila-like dosage compensation mechanism in a reptile lineage.
Marin R., Cortez D., Lamanna F., Pradeepa M.M., Leushkin E., Julien P., Liechti A., Halbert J., Brüning T., Mössinger K. et al., 2017/12. Genome research, 27 (12) pp. 1974-1987. Peer-reviewed.
 
Sex-biased microRNA expression in mammals and birds reveals underlying regulatory mechanisms and a role in dosage compensation.
Warnefors M., Mössinger K., Halbert J., Studer T., VandeBerg J.L., Lindgren I., Fallahshahroudi A., Jensen P., Kaessmann H., 2017/12. Genome research, 27 (12) pp. 1961-1973. Peer-reviewed.
Conserved microRNA editing in mammalian evolution, development and disease.
Warnefors M., Liechti A., Halbert J., Valloton D., Kaessmann H., 2014. Genome Biology, 15 (6) pp. R83.
 
Expression in yeast links field polymorphisms in PfATP6 to in vitro artemisinin resistance and identifies new inhibitor classes.
Pulcini S., Staines H.M., Pittman J.K., Slavic K., Doerig C., Halbert J., Tewari R., Shah F., Avery M.A., Haynes R.K. et al., 2013/08. Journal of Infectious Diseases, 208 (3) pp. 468-478.
 
Global kinomic and phospho-proteomic analyses of the human malaria parasite Plasmodium falciparum.
Solyakov L., Halbert J., Alam M.M., Semblat J.P., Dorin-Semblat D., Reininger L., Bottrill A.R., Mistry S., Abdi A., Fennell C. et al., 2011/11. Nature Communications, 2 p. 565.
 
Two nucleus-localized CDK-like kinases with crucial roles for malaria parasite erythrocytic replication are involved in phosphorylation of splicing factor.
Agarwal S., Kern S., Halbert J., Przyborski J.M., Baumeister S., Dandekar T., Doerig C., Pradel G., 2011/05. Journal of Cellular Biochemistry, 112 (5) pp. 1295-1310.
 
A Plasmodium falciparum transcriptional cyclin-dependent kinase-related kinase with a crucial role in parasite proliferation associates with histone deacetylase activity.
Halbert J., Ayong L., Equinet L., Le Roch K., Hardy M., Goldring D., Reininger L., Waters N., Chakrabarti D., Doerig C., 2010/06. Eukaryotic Cell, 9 (6) pp. 952-959.
 
Malaria: targeting parasite and host cell kinomes.
Doerig C., Abdi A., Bland N., Eschenlauer S., Dorin-Semblat D., Fennell C., Halbert J., Holland Z., Nivez M.P., Semblat J.P. et al., 2010/03. Biochimica et Biophysica Acta, 1804 (3) pp. 604-612.
 
Functional characterization of both MAP kinases of the human malaria parasite Plasmodium falciparum by reverse genetics.
Dorin-Semblat D., Quashie N., Halbert J., Sicard A., Doerig C., Peat E., Ranford-Cartwright L., Doerig C., 2007/09. Molecular Microbiology, 65 (5) pp. 1170-1180.
 
Host genotype by parasite genotype interactions underlying the resistance of anopheline mosquitoes to Plasmodium falciparum.
Lambrechts L., Halbert J., Durand P., Gouagna L.C., Koella J.C., 2005/01. Malaria Journal, 4 p. 3.
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