Fabienne Tacchini-Cottier

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97 publications

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TLR2 Signaling in Skin Nonhematopoietic Cells Induces Early Neutrophil Recruitment in Response to Leishmania major Infection.
Ronet C., Passelli K., Charmoy M., Scarpellino L., Myburgh E., Hauyon La Torre Y., Turco S., Mottram J.C., Fasel N., Luther S.A. et al., 2019/06. The Journal of investigative dermatology, 139 (6) pp. 1318-1328. Peer-reviewed.
Deletion of Interleukin-4 Receptor Alpha-Responsive Keratinocytes in BALB/c Mice Does Not Alter Susceptibility to Cutaneous Leishmaniasis.
Govender M., Hurdayal R., Martinez-Salazar B., Gqada K., Pillay S., Gcanga L., Passelli K., Nieuwenhuizen N.E., Tacchini-Cottier F., Guler R. et al., 2018/12. Infection and immunity, 86 (12) pp. e00710-18. Peer-reviewed.
Neutrophils enhance early Trypanosoma brucei infection onset.
Caljon G., Mabille D., Stijlemans B., De Trez C., Mazzone M., Tacchini-Cottier F., Malissen M., A Van Ginderachter J., Magez S., De Baetselier P. et al., 2018. Scientific Reports, 8 (1) p. 11203. Peer-reviewed.
Resistance of Leishmania (Viannia) Panamensis to Meglumine Antimoniate or Miltefosine Modulates Neutrophil Effector Functions.
Regli I.B., Fernández O.L., Martínez-Salazar B., Gómez M.A., Saravia N.G., Tacchini-Cottier F., 2018. Frontiers in Immunology, 9 p. 3040. Peer-reviewed.
Survival Mechanisms Used by Some Leishmania Species to Escape Neutrophil Killing
Regli I.B., Passelli K., Hurrell B.P., Tacchini-Cottier F., 2017/11/16. Frontiers in Immunology, 8 p. 1558. Peer-reviewed.
Targeting Prolyl-tRNA Synthetase to Accelerate Drug Discovery against Malaria, Leishmaniasis, Toxoplasmosis, Cryptosporidiosis, and Coccidiosis.
Jain V., Yogavel M., Kikuchi H., Oshima Y., Hariguchi N., Matsumoto M., Goel P., Touquet B., Jumani R.S., Tacchini-Cottier F. et al., 2017/10/03. Structure, 25 (10) pp. 1495-1505.e6. Peer-reviewed.
Frontline Science: Leishmania mexicana amastigotes can replicate within neutrophils.
Hurrell B.P., Beaumann M., Heyde S., Regli I.B., Müller A.J., Tacchini-Cottier F., 2017. Journal of Leukocyte Biology, 102 (5) pp. 1187-1198. Peer-reviewed.
IL-4Rα Signaling in Keratinocytes and Early IL-4 Production Are Dispensable for Generating a Curative T Helper 1 Response in Leishmania major-Infected C57BL/6 Mice.
Descatoire M., Hurrell B.P., Govender M., Passelli K., Martinez-Salazar B., Hurdayal R., Brombacher F., Guler R., Tacchini-Cottier F., 2017. Frontiers in Immunology, 8 p. 1265. Peer-reviewed.
Notch regulates Th17 differentiation and controls trafficking of IL-17 and metabolic regulators within Th17 cells in a context-dependent manner.
Coutaz M., Hurrell B.P., Auderset F., Wang H., Siegert S., Eberl G., Ho P.C., Radtke F., Tacchini-Cottier F., 2016/12/15. Scientific reports, 6 p. 39117. Peer-reviewed.
Notch Signaling Regulates the Homeostasis of Tissue-Restricted Innate-like T Cells.
Chennupati V., Koch U., Coutaz M., Scarpellino L., Tacchini-Cottier F., Luther S.A., Radtke F., Zehn D., MacDonald H.R., 2016/08/01. Journal of immunology, 197 (3) pp. 771-782. Peer-reviewed.
Different Leishmania Species Drive Distinct Neutrophil Functions.
Hurrell B.P., Regli I.B., Tacchini-Cottier F., 2016. Trends in Parasitology, 32 (5) pp. 392-401. Peer-reviewed.
NLRP12 is a neutrophil-specific, negative regulator of in vitro cell migration but does not modulate LPS- or infection-induced NF-κB or ERK signalling.
Zamoshnikova A., Groß C.J., Schuster S., Chen K.W., Wilson A., Tacchini-Cottier F., Schroder K., 2016. Immunobiology, 221 (2) pp. 341-346.
The Nlrp3 inflammasome, IL-1β, and neutrophil recruitment are required for susceptibility to a nonhealing strain of Leishmania major in C57BL/6 mice.
Charmoy M., Hurrell B.P., Romano A., Lee S.H., Ribeiro-Gomes F., Riteau N., Mayer-Barber K., Tacchini-Cottier F., Sacks D.L., 2016. European Journal of Immunology, 46 (4) pp. 897-911.
Exposure to Leishmania braziliensis triggers neutrophil activation and apoptosis.
Falcão S.A., Weinkopff T., Hurrell B.P., Celes F.S., Curvelo R.P., Prates D.B., Barral A., Borges V.M., Tacchini-Cottier F., de Oliveira C.I., 2015. PLoS Neglected Tropical Diseases, 9 (3) pp. e0003601.
Rapid Sequestration of Leishmania mexicana by Neutrophils Contributes to the Development of Chronic Lesion.
Hurrell B.P., Schuster S., Grün E., Coutaz M., Williams R.A., Held W., Malissen B., Malissen M., Yousefi S., Simon H.U. et al., 2015. PLoS Pathogens, 11 (5) pp. e1004929.
A scoring method to standardize lesion monitoring following intra-dermal infection of Leishmania parasites in the murine ear
Schuster S., Hartley M.A., Tacchini-Cottier F., Ronet C., 2014. Frontiers in Cellular and Infection Microbiology, 4 p. 67.
Activated group 3 innate lymphoid cells promote T-cell-mediated immune responses.
von Burg N., Chappaz S., Baerenwaldt A., Horvath E., Bose Dasgupta S., Ashok D., Pieters J., Tacchini-Cottier F., Rolink A., Acha-Orbea H. et al., 2014. Proceedings of the National Academy of Sciences of the United States of America, 111 (35) pp. 12835-12840.
Blocking Junctional Adhesion Molecule C Enhances Dendritic Cell Migration and Boosts the Immune Responses against Leishmania major.
Ballet R., Emre Y., Jemelin S., Charmoy M., Tacchini-Cottier F., Imhof B.A., 2014. PLoS Pathogens, 10 (12) pp. e1004550.
Cross-presenting dendritic cells are required for control of Leishmania major infection.
Ashok D., Schuster S., Ronet C., Rosa M., Mack V., Lavanchy C., Marraco S.F., Fasel N., Murphy K.M., Tacchini-Cottier F. et al., 2014. European Journal of Immunology, 44 (5) pp. 1422-1432.
Repeated exposure to Lutzomyia intermedia sand fly saliva induces local expression of interferon-inducible genes both at the site of injection in mice and in human blood.
Weinkopff T., de Oliveira C.I., de Carvalho A.M., Hauyon-La Torre Y., Muniz A.C., Miranda J.C., Barral A., Tacchini-Cottier F., 2014. PLoS Neglected Tropical Diseases, 8 (1) pp. e2627.
Specific fibroblastic niches in secondary lymphoid organs orchestrate distinct Notch-regulated immune responses.
Fasnacht N., Huang H.Y., Koch U., Favre S., Auderset F., Chai Q., Onder L., Kallert S., Pinschewer D.D., MacDonald H.R. et al., 2014. Journal of Experimental Medicine, 211 (11) pp. 2265-2279.
Trapping of naive lymphocytes triggers rapid growth and remodeling of the fibroblast network in reactive murine lymph nodes.
Yang C.Y., Vogt T.K., Favre S., Scarpellino L., Huang H.Y., Tacchini-Cottier F., Luther S.A., 2014. Proceedings of the National Academy of Sciences of the United States of America, 111 (1) pp. E109-E118.
TREM-1 deficiency can attenuate disease severity without affecting pathogen clearance.
Weber B., Schuster S., Zysset D., Rihs S., Dickgreber N., Schürch C., Riether C., Siegrist M., Schneider C., Pawelski H. et al., 2014. PLoS Pathogens, 10 (1) pp. e1003900.
Arginase activity - a marker of disease status in patients with visceral leishmaniasis in ethiopia.
Abebe T., Takele Y., Weldegebreal T., Cloke T., Closs E., Corset C., Hailu A., Hailu W., Sisay Y., Corware K. et al., 2013. PLoS Neglected Tropical Diseases, 7 (3) pp. e2134.
Arginase activity in the blood of patients with visceral leishmaniasis and HIV infection.
Takele Y., Abebe T., Weldegebreal T., Hailu A., Hailu W., Hurissa Z., Ali J., Diro E., Sisay Y., Cloke T. et al., 2013. PLoS Neglected Tropical Diseases, 7 (1) pp. e1977.
Crosstalk between neutrophils and dendritic cells: a context-dependent process.
Schuster S., Hurrell B., Tacchini-Cottier F., 2013. Journal of Leukocyte Biology, 94 (4) pp. 671-675.
Notch signaling regulates follicular helper T cell differentiation.
Auderset F., Schuster S., Fasnacht N., Coutaz M., Charmoy M., Koch U., Favre S., Wilson A., Trottein F., Alexander J. et al., 2013. Journal of Immunology, 191 (5) pp. 2344-2350.
Regulation of innate and adaptive immunity by Notch.
Radtke F., Macdonald H.R., Tacchini-Cottier F., 2013. Nature Reviews. Immunology, 13 (6) pp. 427-437.
Role of Toll-Like Receptor 9 Signaling in Experimental Leishmania braziliensis Infection.
Weinkopff T., Mariotto A., Simon G., Hauyon-La Torre Y., Auderset F., Schuster S., Zangger H., Fasel N., Barral A., Tacchini-Cottier F., 2013. Infection and Immunity, 81 (5) pp. 1575-1584.
Differential peptide binding to CD40 evokes counteractive responses.
Khan S., Alonso L., Roduit C., Bandyopadhyay S., Singh S., Saha S., Tacchini-Cottier F., Roy S., Dietler G., Kasas S. et al., 2012. Human Immunology, 73 (5) pp. 465-469.
Does T Helper Differentiation Correlate with Resistance or Susceptibility to Infection with L. major? Some Insights From the Murine Model.
Tacchini-Cottier F., Weinkopff T., Launois P., 2012. Frontiers in Immunology, 3 p. 32.
Environmental and T cell-intrinsic factors limit the expansion of neonatal follicular T helper cells but may be circumvented by specific adjuvants.
Mastelic B., Kamath A.T., Fontannaz P., Tougne C., Rochat A.F., Belnoue E., Combescure C., Auderset F., Lambert P.H., Tacchini-Cottier F. et al., 2012. Journal of Immunology, 189 (12) pp. 5764-5772.
Local increase of arginase activity in lesions of patients with cutaneous leishmaniasis in ethiopia.
Abebe T., Hailu A., Woldeyes M., Mekonen W., Bilcha K., Cloke T., Fry L., Seich Al Basatena N.K., Corware K., Modolell M. et al., 2012. PLoS Neglected Tropical Diseases, 6 (6) pp. e1684.
Redundant Notch1 and Notch2 signaling is necessary for IFNγ secretion by T helper 1 cells during infection with Leishmania major.
Auderset F., Schuster S., Coutaz M., Koch U., Desgranges F., Merck E., MacDonald H.R., Radtke F., Tacchini-Cottier F., 2012. PLoS Pathogens, 8 (3) pp. e1002560.
The role of Notch in the differentiation of CD4⁺ T helper cells.
Auderset F., Coutaz M., Tacchini-Cottier F., 2012. Current Topics in Microbiology and Immunology, 360 pp. 115-134.
Amine-reactive OVA multimers for auto-vaccination against cytokines and other mediators: perspectives illustrated for GCP-2 in L. major infection.
Uyttenhove C., Marillier R.G., Tacchini-Cottier F., Charmoy M., Caspi R.R., Damsker J.M., Goriely S., Su D., Van Damme J., Struyf S. et al., 2011. Journal of Leukocyte Biology, 89 (6) pp. 1001-1007.
Neutrophils in infectious diseases
Tacchini-Cottier F., van Zandbergen G. (eds.), 2011. 79, Bentham e Books.
Role of neutrophils in the early shaping of the Leishmania major specific immune response in experimental murine cutaneous Leishmaniasis
Charmoy M, Milon G., Tacchini-Cottier F., 2011. pp. 49-58 dans Tacchini-Cottier F., van Zandbergen G. (eds.) Neutrophils in infectious diseases, Bentham e Books.
Neutrophil-derived CCL3 is essential for the rapid recruitment of dendritic cells to the site of Leishmania major inoculation in resistant mice.
Charmoy M., Brunner-Agten S., Aebischer D., Auderset F., Launois P., Milon G., Proudfoot A.E., Tacchini-Cottier F., 2010. PLoS Pathogens, 6 (2) pp. e1000755.
Regulatory B cells shape the development of Th2 immune responses in BALB/c mice infected with Leishmania major through IL-10 production.
Ronet C., Hauyon-La Torre Y., Revaz-Breton M., Mastelic B., Tacchini-Cottier F., Louis J., Launois P., 2010. Journal of Immunology, 184 (2) pp. 886-894. Peer-reviewed.
The MyD88 protein 88 pathway is differently involved in immune responses induced by distinct substrains of Leishmania major.
Revaz-Breton M., Ronet C., Ives A., Torre Y.H., Masina S., Tacchini-Cottier F., Launois P., 2010. European Journal of Immunology, 40 (6) pp. 1697-1707. Peer-reviewed.
The prominent role of neutrophils during the initial phase of infection by Leishmania parasites.
Charmoy M., Auderset F., Allenbach C., Tacchini-Cottier F., 2010. Journal of Biomedicine and Biotechnology, 2010 p. 719361. Peer-reviewed.
A new function of the Fas-FasL pathway in macrophage activation.
Chakour R., Allenbach C., Desgranges F., Charmoy M., Mauel J., Garcia I., Launois P., Louis J., Tacchini-Cottier F., 2009. Journal of Leukocyte Biology, 86 (1) pp. 81-90. Peer-reviewed.
Dynamic regulation of notch 1 and notch 2 surface expression during T cell development and activation revealed by novel monoclonal antibodies.
Fiorini E., Merck E., Wilson A., Ferrero I., Jiang W., Koch U., Auderset F., Laurenti E., Tacchini-Cottier F., Pierres M. et al., 2009. Journal of immunology, 183 (11) pp. 7212-7222. Peer-reviewed.
In leishmaniasis due to Leishmania guyanensis infection, distinct intralesional interleukin-10 and Foxp3 mRNA expression are associated with unresponsiveness to treatment.
Bourreau E., Ronet C., Darsissac E., Lise M.C., Marie D.S., Clity E., Tacchini-Cottier F., Couppie P., Launois P., 2009. The Journal of Infectious Diseases, 199 (4) pp. 576-579. Peer-reviewed.
Intralesional regulatory T-cell suppressive function during human acute and chronic cutaneous leishmaniasis due to Leishmania guyanensis.
Bourreau E., Ronet C., Darcissac E., Lise M.C., Sainte Marie D., Clity E., Tacchini-Cottier F., Couppie P., Launois P., 2009. Infection and Immunity, 77 (4) pp. 1465-1474. Peer-reviewed.
An essential role for transmembrane TNF in the resolution of the inflammatory lesion induced by Leishmania major infection.
Allenbach C., Launois P., Mueller C., Tacchini-Cottier F., 2008. European Journal of Immunology, 38 (3) pp. 720-731.
Cutaneous leishmaniasis: progress towards a vaccine.
Launois P., Tacchini-Cottier F., Kieny M.P., 2008. Expert Review of Vaccines, 7 (8) pp. 1277-1287.
Host responses to infections with Leishmania
Tacchini-Cottier F., Launois P., 2008. pp. 263-280 dans Myler P.J., Fasel N. (eds.) Leishmania : after the Genome chap. 12, Caister Academic Press.
Leishmania major-specific B cells are necessary for Th2 cell development and susceptibility to L. major LV39 in BALB/c mice.
Ronet C., Voigt H., Himmelrich H., Doucey M.A., Hauyon-La Torre Y., Revaz-Breton M., Tacchini-Cottier F., Bron C., Louis J., Launois P., 2008. Journal of Immunology, 180 (7) pp. 4825-4835. Peer-reviewed.
Neutrophils contribute to development of a protective immune response during onset of infection with Leishmania donovani.
McFarlane E., Perez C., Charmoy M., Allenbach C., Carter K.C., Alexander J., Tacchini-Cottier F., 2008. Infection and immunity, 76 (2) pp. 532-541. Peer-reviewed.
IL-10 producing CD8+ T cells in human infection with Leishmania guyanensis.
Bourreau E., Ronet C., Couppié P., Sainte-Marie D., Tacchini-Cottier F., Launois P., 2007. Microbes and Infection, 9 (8) pp. 1034-1041.
Interleukin-12p40 overexpression promotes interleukin-12p70 and interleukin-23 formation but does not affect bacille Calmette-Guérin and Mycobacterium tuberculosis clearance.
Olleros M.L., Vesin D., Martinez-Soria E., Allenbach C., Tacchini-Cottier F., Pache J.C., Marchal G., Rahman J., Fernández C., Izui S. et al., 2007. Immunology, 122 (3) pp. 350-361.
Leishmania major induces distinct neutrophil phenotypes in mice that are resistant or susceptible to infection.
Charmoy M., Megnekou R., Allenbach C., Zweifel C., Perez C., Monnat K., Breton M., Ronet C., Launois P., Tacchini-Cottier F., 2007. Journal of Leukocyte Biology, 82 (2) pp. 288-299.
Selective expression of the V beta 14 T cell receptor on Leishmania guyanensis--specific CD8+ T cells during human infection.
Kariminia A., Bourreau E., Ronet C., Couppie P., Sainte-Marie D., Tacchini-Cottier F., Launois P., 2007. Journal of Infectious Diseases, 195 (5) pp. 739-747.
LACK-reactive CD4+ T cells require autocrine IL-2 to mediate susceptibility to Leishmania major.
Gumy A., Aseffa A., Rachinel N., Breton M., Otten L., Tacchini-Cottier F., Röcken M., Doyen N., Acha-Orbea H., Locksley R.M. et al., 2006. European Journal of Immunology, 36 (6) pp. 1465-1473. Peer-reviewed.
Macrophages induce neutrophil apoptosis through membrane TNF, a process amplified by Leishmania major.
Allenbach C., Zufferey C., Perez C., Launois P., Mueller C., Tacchini-Cottier F., 2006. Journal of Immunology, 176 (11) pp. 6656-6664.
Pregnancy impairs resistance of C57BL/6 mice to Leishmania major infection.
Arinola O.G., Louis J.S., Tacchini-Cottier F., Aseffa A., Salimonu L.S., 2005. African Journal of Medicine and Medical Sciences, 34 (1) pp. 65-70.
Transforming growth factor beta 1 production by CD4+ CD25+ regulatory T cells in peripheral blood mononuclear cells from healthy subjects stimulated with Leishmania guyanensis.
Kariminia A., Bourreau E., Pascalis H., Couppié P., Sainte-Marie D., Tacchini-Cottier F., Launois P., 2005. Infection and Immunity, 73 (9) pp. 5908-5914.
Inhibition of inducible nitric oxide synthase protects against liver injury induced by mycobacterial infection and endotoxins.
Guler R., Olleros M.L., Vesin D., Parapanov R., Vesin C., Kantengwa S., Rubbia-Brandt L., Mensi N., Angelillo-Scherrer A., Martinez-Soria E. et al., 2004. Journal of Hepatology, 41 (5) pp. 773-781. Peer-reviewed.
Interleukin-4 (IL-4) and interferon-gamma (IFN-gamma) in pregnant C57BL/6 mice infected with L. major at different gestational periods.
Arinola O.G., Louis J., Tacchini-Cottier F., Aseffa A., Salimonu L.S., 2004. West African Journal of Medicine, 23 (3) pp. 202-207.
Notch1 expression on T cells is not required for CD4+ T helper differentiation.
Tacchini-Cottier F., Allenbach C., Otten L.A., Radtke F., 2004. European Journal of Immunology, 34 (6) pp. 1588-1596.
Both the Fas ligand and inducible nitric oxide synthase are needed for control of parasite replication within lesions in mice infected with Leishmania major whereas the contribution of tumor necrosis factor is minimal.
Chakour R., Guler R., Bugnon M., Allenbach C., Garcia I., Mauël J., Louis J., Tacchini-Cottier F., 2003. Infection and Immunity, 71 (9) pp. 5287-5295.
Deregulated MHC class II transactivator expression leads to a strong Th2 bias in CD4+ T lymphocytes.
Otten L.A., Tacchini-Cottier F., Lohoff M., Annunziato F., Cosmi L., Scarpellino L., Louis J., Steimle V., Reith W., Acha-Orbea H., 2003. Journal of Immunology, 170 (3) pp. 1150-1157.
Parasitic diseases, with an emphasis on experimental cutaneous Leishmaniasis
Launois P., Voigt H., Gumy A., Aseffa A., Tacchini-Cottier F., Röcken M., Louis J.A., 2003. pp. 165-177 dans Bloom B.R., Lambert P.H. (eds.) The Vaccine Book, Academic Press.
Mouse CD11c(+) B220(+) Gr1(+) plasmacytoid dendritic cells develop independently of the T-cell lineage.
Ferrero I., Held W., Wilson A., Tacchini-Cottier F., Radtke F., MacDonald H.R., 2002. Blood, 100 (8) pp. 2852-2857. Peer-reviewed.
The early IL-4 response to Leishmania major and the resulting Th2 cell maturation steering progressive disease in BALB/c mice are subject to the control of regulatory CD4+CD25+ T cells.
Aseffa A., Gumy A., Launois P., MacDonald H.R., Louis J.A., Tacchini-Cottier F., 2002. Journal of Immunology, 169 (6) pp. 3232-3241.
An immunomodulatory function for neutrophils during the induction of a CD4+ Th2 response in BALB/c mice infected with Leishmania major.
Tacchini-Cottier F., Zweifel C., Belkaid Y., Mukankundiye C., Vasei M., Launois P., Milon G., Louis J.A., 2000. Journal of Immunology, 165 (5) pp. 2628-2636.
Delayed mortality and attenuated thrombocytopenia associated with severe malaria in urokinase- and urokinase receptor-deficient mice.
Piguet P.F., Da Laperrousaz C., Vesin C., Tacchini-Cottier F., Senaldi G., Grau G.E., 2000. Infection and Immunity, 68 (7) pp. 3822-3829.
In BALB/c mice, IL-4 production during the initial phase of infection with Leishmania major is necessary and sufficient to instruct Th2 cell development resulting in progressive disease.
Himmelrich H., Launois P., Maillard I., Biedermann T., Tacchini-Cottier F., Locksley R.M., Röcken M., Louis J.A., 2000. Journal of Immunology, 164 (9) pp. 4819-4825.
A role for lymphotoxin beta receptor in host defense against Mycobacterium bovis BCG infection.
Lucas R., Tacchini-Cottier F., Guler R., Vesin D., Jemelin S., Olleros M.L., Marchal G., Browning J.L., Vassalli P., Garcia I., 1999. European Journal of Immunology, 29 (12) pp. 4002-4010.
New insight into the mechanisms underlying Th2 cell development and susceptibility to Leishmania major in BALB/c mice.
Launois P., Himmelrich H., Tacchini-Cottier F., Milon G., Louis J.A., 1999. Microbes and Infection, 1 (1) pp. 59-64.
Some of the early events underlying Th2 cell maturation and susceptibility to Leishmania major infection in BALB/c mice.
Himmelrich H., Launois P., Tacchini-Cottier F., Louis J.A., 1999. Biological Chemistry, 380 (7-8) pp. 909-914.
Urokinase receptor (uPAR, CD87) is a platelet receptor important for kinetics and TNF-induced endothelial adhesion in mice.
Piguet P.F., Vesin C., Donati Y., Tacchini-Cottier F., Belin D., Barazzone C., 1999. Circulation, 99 (25) pp. 3315-3321.
Anti-leishmania effector functions of CD4+ Th1 cells and early events instructing Th2 cell development and susceptibility to Leishmania major in BALB/c mice.
Louis J.A., Conceiçao-Silva F., Himmelrich H., Tacchini-Cottier F., Launois P., 1998. Advances in Experimental Medicine and Biology, 452 pp. 53-60.
Cytokines in parasitic diseases: the example of cutaneous leishmaniasis.
Launois P., Tacchini-Cottier F., Parra-Lopez C., Louis J.A., 1998. International Reviews of Immunology, 17 (1-4) pp. 157-180.
Regulation of protective immunity against Leishmania major in mice.
Louis J., Himmelrich H., Parra-Lopez C., Tacchini-Cottier F., Launois P., 1998. Current Opinion in Immunology, 10 (4) pp. 459-464.
Role of TNFR1 and TNFR2 in TNF-induced platelet consumption in mice.
Tacchini-Cottier F., Vesin C., Redard M., Buurman W., Piguet P.F., 1998. Journal of Immunology, 160 (12) pp. 6182-6186.
Setting in motion the immune mechanisms underlying genetically determined resistance and susceptibility to infection with Leishmania major.
Launois P., Conceiçao-Silva F., Himmerlich H., Parra-Lopez C., Tacchini-Cottier F., Louis J.A., 1998. Parasite Immunology, 20 (5) pp. 223-230.
The IL-4 rapidly produced in BALB/c mice after infection with Leishmania major down-regulates IL-12 receptor beta 2-chain expression on CD4+ T cells resulting in a state of unresponsiveness to IL-12.
Himmelrich H., Parra-Lopez C., Tacchini-Cottier F., Louis J.A., Launois P., 1998. Journal of Immunology, 161 (11) pp. 6156-6163.
Heat shock and proinflammatory stressors induce differential localization of heat shock proteins in human monocytes.
Mariéthoz E., Jacquier-Sarlin M.R., Multhoff G., Healy A.M., Tacchini-Cottier F., Polla B.S., 1997. Inflammation, 21 (6) pp. 629-642.
Polyoma middle T-induced vascular tumor formation: the role of the plasminogen activator/plasmin system.
Sabapathy K.T., Pepper M.S., Kiefer F., Möhle-Steinlein U., Tacchini-Cottier F., Fetka I., Breier G., Risau W., Carmeliet P., Montesano R. et al., 1997. Journal of Cell Biology, 137 (4) pp. 953-963.
Activated human platelets express beta2 integrin.
Philippeaux M.M., Vesin C., Tacchini-Cottier F., Piguet P.F., 1996. European Journal of Haematology, 56 (3) pp. 130-137.
Hyperoxia induces platelet activation and lung sequestration: an event dependent on tumor necrosis factor-alpha and CD11a.
Barazzone C., Tacchini-Cottier F., Vesin C., Rochat A.F., Piguet P.F., 1996. American Journal of Respiratory Cell and Molecular Biology, 15 (1) pp. 107-114.
Administration of anti-TNF-alpha or anti-CD11a antibodies to normal adult mice decreases lung and bone collagen content: evidence for an effect on platelet consumption.
Piguet P.F., Tacchini-Cottier F., Vesin C., 1995. American Journal of Respiratory Cell and Molecular Biology, 12 (2) pp. 227-231.
Detection of a LFA-1-like epitope on the surface of erythrocytes infected with a strain of Plasmodium falciparum.
Tacchini-Cottier F., Lou J.N., Roberts D.J., Garcia A.M., Grau G.E., 1995. Immunology, 85 (2) pp. 205-213.
Inactivation of E alpha and E beta expression in inbred and wild mice by multiple distinct mutations, some of which predate speciation within Mus species.
Tacchini-Cottier F., Mayer W.E., Begovich A.B., Jones P.P., 1995. International Immunology, 7 (9) pp. 1459-1471.
Cytokines et paludisme
Tacchini-Cottier F., Grau G.E., 1994. Médecine Tropicale, 54 (3) pp. 257-262.
Exposure of monocytes to heat shock does not increase class II expression but modulates antigen-dependent T cell responses.
Mariéthoz E., Tacchini-Cottier F., Jacquier-Sarlin M., Sinclair F., Polla B.S., 1994. International Immunology, 6 (6) pp. 925-930.
Galactose residues in Plasmodium falciparum glycoproteins: incorporation and elimination.
Qazi M.A., Senaldi G., Tacchini-Cottier F., Qazi M.H., Fayyaz-ud-Din D.C., Hoessli D.C., Walker-Nasir E., Nasir-ud-Din , 1994. Biochemical Society Transactions, 22 (3) pp. 362S.
An effector role for platelets in systemic and local lipopolysaccharide-induced toxicity in mice, mediated by a CD11a- and CD54-dependent interaction with endothelium.
Piguet P.F., Vesin C., Ryser J.E., Senaldi G., Grau G.E., Tacchini-Cottier F., 1993. Infection and Immunity, 61 (10) pp. 4182-4187.
TNF-induced microvascular pathology: active role for platelets and importance of the LFA-1/ICAM-1 interaction.
Grau G.E., Tacchini-Cottier F., Vesin C., Milon G., Lou J.N., Piguet P.F., Juillard P., 1993. European Cytokine Network, 4 (6) pp. 415-419.
Toxoplasmic encephalitis.
Grau G.E., Tacchini-Cottier F., Piguet P.F., 1992. Parasitology Today, 8 (11) p. 367.
Evolution of class II genes: role of selection in both the maintenance of polymorphism and the retention of non-expressed alleles.
Jones P.P., Begovich A.B., Tacchini-Cottier F.M., Vu T.H., 1990. Immunologic Research, 9 (3) pp. 200-211.
Molecular defects in the non-expressed H-2 E alpha genes of the f and q haplotypes.
Vu T.H., Begovich A.B., Tacchini-Cottier F.M., Jones P.P., 1989. Journal of Immunology, 142 (8) pp. 2936-2942.
Defective E beta expression in three mouse H-2 haplotypes results from aberrant RNA splicing.
Tacchini-Cottier F.M., Jones P.P., 1988. Journal of Immunology, 141 (10) pp. 3647-3653.
Molecular basis for the defective expression of the mouse Ew17 beta gene.
Vu T.H., Tacchini-Cottier F.M., Day C.E., Begovich A.B., Jones P.P., 1988. Journal of Immunology, 141 (10) pp. 3654-3661.
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