Gilles Willemin

Publications | Phd and Masters theses

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6 publications

2023 | 2020 | 2019 | 2015 | 2013 |
2023
 
Author Correction: Possible association of 16p11.2 copy number variation with altered lymphocyte and neutrophil counts.
Giannuzzi G., Chatron N., Mannik K., Auwerx C., Pradervand S., Willemin G., Hoekzema K., Nuttle X., Chrast J., Sadler M.C. et al., 2023/05/24. NPJ genomic medicine, 8 (1) p. 9. Peer-reviewed.
[DOI][WoS][Pmid][serval:BIB_BB9CACD534D5]
 
Contrasting effects of whole-body and hepatocyte-specific deletion of the RNA polymerase III repressor Maf1 in the mouse.
Willemin G., Mange F., Praz V., Lorrain S., Cousin P., Roger C., Willis I.M., Hernandez N., 2023. Frontiers in molecular biosciences, 10 p. 1297800. Peer-reviewed.
[DOI][WoS][Pmid][serval:BIB_83067C6358E5]
2020
MAF1 is a chronic repressor of RNA polymerase III transcription in the mouse.
Bonhoure N., Praz V., Moir R.D., Willemin G., Mange F., Moret C., Willis I.M., Hernandez N., 2020/07/20. Scientific reports, 10 (1) p. 11956. Peer-reviewed.
[URN][DOI][Pmid][serval:BIB_66A30385CDCC]
2019
 
The Human-Specific BOLA2 Duplication Modifies Iron Homeostasis and Anemia Predisposition in Chromosome 16p11.2 Autism Individuals
Giannuzzi Giuliana, Schmidt Paul J., Porcu Eleonora, Willemin Gilles, Munson Katherine M., Nuttle Xander, Earl Rachel, Chrast Jacqueline, Hoekzema Kendra, Risso Davide et al., 2019/11. The American Journal of Human Genetics, 105 (5) pp. 947-958.
[DOI][WoS][Pmid][serval:BIB_4E12AE891247]
2015
Loss of the RNA polymerase III repressor MAF1 confers obesity resistance.
Bonhoure N., Byrnes A., Moir R.D., Hodroj W., Preitner F., Praz V., Marcelin G., Chua S.C., Martinez-Lopez N., Singh R. et al., 2015. Genes and Development, 29 (9) pp. 934-947.
[URN][DOI][WoS][Pmid][serval:BIB_F415BF94B601]
2013
Major Histocompatibility Class II Pathway Is Not Required for the Development of Nonalcoholic Fatty Liver Disease in Mice.
Willemin G., Roger C., Bauduret A., Minehira K., 2013. International Journal of Endocrinology, 2013 p. 972962.
[URN][DOI][WoS][Pmid][serval:BIB_89E74231738A]
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