Olivier Braissant

Publications | Mémoires et thèses

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107 publications

2023 | 2022 | 2021 | 2020 | 2019 | 2018 | 2017 | 2016 | 2015 | 2014 | 2013 | 2012 | 2011 | 2010 | 2009 | 2008 | 2007 | 2006 | 2005 | 2004 | 2003 | 2002 | 2001 | 1999 | 1998 | 1997 | 1996 | 1995 | 1991 |
Neurometabolic changes in a rat pup model of type C hepatic encephalopathy depend on age at liver disease onset.
Simicic D., Rackayova V., Braissant O., Toso C., Oldani G., Sessa D., McLin V.A., Cudalbu C., 2023/08. Metabolic brain disease, 38 (6) pp. 1999-2012. Peer-reviewed.
PET CMR<sub>glc</sub> mapping and <sup>1</sup>H-MRS show altered glucose uptake and neurometabolic profiles in BDL rats.
Mosso J., Yin T., Poitry-Yamate C., Simicic D., Lepore M., McLin V.A., Braissant O., Cudalbu C., Lanz B., 2022/06/15. Analytical biochemistry, 647 p. 114606. Peer-reviewed.
 
Obituary for Claude Bachmann, MD (1941–2022).
Batshaw ML, Braissant O., Dionisi-Vici C., Steinmann B., Superti-Furga A., 2022/05. Journal of Inherited Metabolic Disease, 45 (3) pp. 657-658. Peer-reviewed.
Corrigendum to "Central nervous system and systemic oxidative stress interplay with inflammation in a bile duct ligation rat model of type C hepatic encephalopathy" [Free Radical Biol. Med. 178 (2022) 295-307 ISSN 0891-5849].
Pierzchala K., Simicic D., Sienkiewicz A., Sessa D., Mitrea S., Braissant O., McLin V.A., Gruetter R., Cudalbu C., 2022/03. Free radical biology & medicine, 182 p. 289. Peer-reviewed.
 
Creatine transporter-deficient rat model shows motor dysfunction, cerebellar alterations, and muscle creatine deficiency without muscle atrophy.
Duran-Trio L., Fernandes-Pires G., Grosse J., Soro-Arnaiz I., Roux-Petronelli C., Binz P.A., De Bock K., Cudalbu C., Sandi C., Braissant O., 2022/03. Journal of inherited metabolic disease, 45 (2) pp. 278-291. Peer-reviewed.
 
Central nervous system and systemic oxidative stress interplay with inflammation in a bile duct ligation rat model of type C hepatic encephalopathy.
Pierzchala K., Simicic D., Sienkiewicz A., Sessa D., Mitrea S., Braissant O., McLin V.A., Gruetter R., Cudalbu C., 2022/01. Free radical biology & medicine, 178 pp. 295-307. Peer-reviewed.
Current and potential new treatment strategies for creatine deficiency syndromes.
Fernandes-Pires G., Braissant O., 2022/01. Molecular genetics and metabolism, 135 (1) pp. 15-26. Peer-reviewed.
 
Disorders of creatine metabolism
Stöckler-Ipsiroglu Sylvia, Braissant Olivier, Schulze Andreas, 2022. dans Blau Nenad, Dionisi Vici Carlo, Ferreira Carlos R., Vianey-Saban Christine, van Karnebeek Clara D. M. (eds.) Physician’s Guide to the Diagnosis, Treatment, and Follow-Up of Inherited Metabolic Diseases, Springer International Publishing.
 
A knock-in rat model unravels acute and chronic renal toxicity in glutaric aciduria type I.
Gonzalez Melo M., Fontana A.O., Viertl D., Allenbach G., Prior J.O., Rotman S., Feichtinger R.G., Mayr J.A., Costanzo M., Caterino M. et al., 2021/12. Molecular genetics and metabolism, 134 (4) pp. 287-300. Peer-reviewed.
 
Probiotics combined with rifaximin influence the neurometabolic changes in a rat model of type C HE.
Flatt E., McLin V.A., Braissant O., Pierzchala K., Mastromarino P., Mitrea S.O., Sessa D., Gruetter R., Cudalbu C., 2021/09/09. Scientific reports, 11 (1) p. 17988. Peer-reviewed.
Classical homocystinuria, is it safe to exercise?
Tankeu A.T., Van Winckel G., Campos-Xavier B., Braissant O., Pedro R., Superti-Furga A., Amati F., Tran C., 2021/06. Molecular genetics and metabolism reports, 27 p. 100746. Peer-reviewed.
 
The first knock-in rat model for glutaric aciduria type I allows further insights into pathophysiology in brain and periphery.
Gonzalez Melo M., Remacle N., Cudré-Cung H.P., Roux C., Poms M., Cudalbu C., Barroso M., Gersting S.W., Feichtinger R.G., Mayr J.A. et al., 2021/06. Molecular genetics and metabolism, 133 (2) pp. 157-181. Peer-reviewed.
 
Magnetic resonance spectroscopy in the rodent brain: Experts' consensus recommendations.
Lanz B., Abaei A., Braissant O., Choi I.Y., Cudalbu C., Henry P.G., Gruetter R., Kara F., Kantarci K., Lee P. et al., 2021/05/31. NMR in biomedicine, 34 (5) pp. e4325. Peer-reviewed.
 
Late post-natal neurometabolic development in healthy male rats using <sup>1</sup> H and <sup>31</sup> P magnetic resonance spectroscopy.
Račkayová V., Simicic D., Donati G., Braissant O., Gruetter R., McLin V.A., Cudalbu C., 2021/05. Journal of neurochemistry, 157 (3) pp. 508-519. Peer-reviewed.
Probiotics improve the neurometabolic profile of rats with chronic cholestatic liver disease.
Rackayová V., Flatt E., Braissant O., Grosse J., Capobianco D., Mastromarino P., McMillin M., DeMorrow S., McLin V.A., Cudalbu C., 2021/01/26. Scientific reports, 11 (1) p. 2269. Peer-reviewed.
A new rat model of creatine transporter deficiency reveals behavioral disorder and altered brain metabolism.
Duran-Trio L., Fernandes-Pires G., Simicic D., Grosse J., Roux-Petronelli C., Bruce S.J., Binz P.A., Sandi C., Cudalbu C., Braissant O., 2021/01/15. Scientific reports, 11 (1) p. 1636. Peer-reviewed.
 
The use of 68Ga-EDTA PET allows detecting progressive decline of renal function in rats.
Fontana A.O., Gonzalez Melo M., Allenbach G., Georgantas C., Wang R., Braissant O., Barbey F., Prior J.O., Ballhausen D., Viertl D., 2021. American journal of nuclear medicine and molecular imaging, 11 (6) pp. 519-528. Peer-reviewed.
Longitudinal osmotic and neurometabolic changes in young rats with chronic cholestatic liver disease.
Rackayova V., Braissant O., Rougemont A.L., Cudalbu C., McLin V.A., 2020/05/05. Scientific reports, 10 (1) p. 7536. Peer-reviewed.
 
Argininosuccinate neurotoxicity and prevention by creatine in argininosuccinate lyase deficiency: An in vitro study in rat three-dimensional organotypic brain cell cultures.
Diez-Fernandez C., Hertig D., Loup M., Diserens G., Henry H., Vermathen P., Nuoffer J.M., Häberle J., Braissant O., 2019/11. Journal of inherited metabolic disease, 42 (6) pp. 1077-1087. Peer-reviewed.
 
Reply to: "Magnetic resonance spectroscopy: A surrogate marker of hepatic encephalopathy?"
McLin V.A., Braissant O., Cudalbu C., 2019/11. Journal of hepatology, 71 (5) p. 1057. Peer-reviewed.
 
Longitudinal neurometabolic changes in the hippocampus of a rat model of chronic hepatic encephalopathy.
Braissant O., Rackayová V., Pierzchala K., Grosse J., McLin V.A., Cudalbu C., 2019/09. Journal of hepatology, 71 (3) pp. 505-515. Peer-reviewed.
 
Ammonium accumulation and chemokine decrease in culture media of Gcdh<sup>-/-</sup> 3D reaggregated brain cell cultures.
Cudré-Cung H.P., Remacle N., do Vale-Pereira S., Gonzalez M., Henry H., Ivanisevic J., Schmiesing J., Mühlhausen C., Braissant O., Ballhausen D., 2019/04. Molecular genetics and metabolism, 126 (4) pp. 416-428. Peer-reviewed.
 
Are heterozygous carriers for hereditary fructose intolerance predisposed to metabolic disturbances when exposed to fructose?
Debray F.G., Damjanovic K., Rosset R., Mittaz-Crettol L., Roux C., Braissant O., Barbey F., Bonafé L., De Bandt J.P., Tappy L. et al., 2018/08/01. The American journal of clinical nutrition, 108 (2) pp. 292-299. Peer-reviewed.
 
New in vitro model derived from brain-specific Mut-/- mice confirms cerebral ammonium accumulation in methylmalonic aciduria.
Remacle N., Forny P., Cudré-Cung H.P., Gonzalez-Melo M., do Vale-Pereira S., Henry H., Teav T., Gallart-Ayala H., Braissant O., Baumgartner M. et al., 2018/08. Molecular genetics and metabolism, 124 (4) pp. 266-277. Peer-reviewed.
 
Creatine in the central nervous system: From magnetic resonance spectroscopy to creatine deficiencies.
Rackayova V., Cudalbu C., Pouwels PJW, Braissant O., 2017/07/15. Analytical biochemistry, 529 pp. 144-157. Peer-reviewed.
 
MRS studies of neuroenergetics and glutamate/glutamine exchange in rats: Extensions to hyperammonemic models.
Lanz B., Rackayova V., Braissant O., Cudalbu C., 2017/07/15. Analytical biochemistry, 529 pp. 245-269. Peer-reviewed.
 
Immunolocalization of glutaryl-CoA dehydrogenase (GCDH) in adult and embryonic rat brain and peripheral tissues.
Braissant O., Jafari P., Remacle N., Cudré-Cung H.P., Do Vale Pereira S., Ballhausen D., 2017/02/20. Neuroscience, 343 pp. 355-363. Peer-reviewed.
 
Creatine deficiency syndromes
Schulze A, Braissant O, 2017. pp. 181-190 dans Sarafoglou K, Hoffmann GG, Roth K (eds.) Pediatric Endocrinology and Inborn Errors of Metabolism, McGraw-Hill Education.
 
1H and 31P magnetic resonance spectroscopy in a rat model of chronic hepatic encephalopathy: in vivo longitudinal measurements of brain energy metabolism.
Rackayova V., Braissant O., McLin V.A., Berset C., Lanz B., Cudalbu C., 2016/12. Metabolic brain disease, 31 (6) pp. 1303-1314. Peer-reviewed.
Ammonium accumulation is a primary effect of 2-methylcitrate exposure in an in vitro model for brain damage in methylmalonic aciduria.
Cudré-Cung H.P., Zavadakova P., do Vale-Pereira S., Remacle N., Henry H., Ivanisevic J., Tavel D., Braissant O., Ballhausen D., 2016/09. Molecular genetics and metabolism, 119 (1-2) pp. 57-67. Peer-reviewed.
 
Creatine synthesis and exchanges between brain cells: What can be learned from human creatine deficiencies and various experimental models?
Hanna-El-Daher L., Braissant O., 2016/08. Amino acids, 48 (8) pp. 1877-1895. Peer-reviewed.
Ammonia toxicity to the brain
Braissant O, 2015/03/27. pp. 40-53 dans Haeberle J (eds.) Current Approach to Hyperammonemia, Future Medicine Ltd..
Mild guanidinoacetate increase under partial guanidinoacetate methyltransferase deficiency strongly affects brain cell development.
Hanna-El-Daher L., Béard E., Henry H., Tenenbaum L., Braissant O., 2015. Neurobiology of Disease, 79 pp. 14-27. Peer-reviewed.
 
Creatine disorders
Stöckler S, Braissant O, Schulze A, 2014. pp. 529-540 dans Blau N, Duran M, Gibson KM, Dionisi-Vici C (eds.) Physician's guide to the diagnosis, treatment, and follow-up of inherited metabolic diseases chap. 32, Springer.
 
GAMT deficiency: 20 years of a treatable inborn error of metabolism.
Braissant O., 2014. Molecular Genetics and Metabolism, 111 (1) pp. 1-3.
 
The long non-coding RNA NEAT1 is increased in IUGR placentas, leading to potential new hypotheses of IUGR origin/development.
Gremlich S., Damnon F., Reymondin D., Braissant O., Schittny J.C., Baud D., Gerber S., Roth-Kleiner M., 2014. Placenta, 35 (1) pp. 44-49. Peer-reviewed.
 
Altered expression and posttranslational modification of neurofilaments in methylmalonic aciduria
Cung H.P., Zavadakova P., Braissant O., Ballhausen D., 2013. pp. S173 dans ICIEM 2013, 12th International Congress of Inborn Errors of Metabolism, Journal of Inherited Metabolic Diseases.
Ammonia toxicity to the brain.
Braissant O., McLin V.A., Cudalbu C., 2013. Journal of Inherited Metabolic Disease, 36 (4) pp. 595-612. Peer-reviewed.
Ammonium accumulation and cell death in a rat 3D brain cell model of glutaric aciduria type I.
Jafari P., Braissant O., Zavadakova P., Henry H., Bonafé L., Ballhausen D., 2013. Plos One, 8 (1) pp. e53735. Peer-reviewed.
Brain damage in methylmalonic aciduria: 2-methylcitrate induces cerebral ammonium accumulation and apoptosis in 3D organotypic brain cell cultures
Jafari P, Braissant O, Zavadakova P, Henry H, Bonafé L, Ballhausen D, 2013. Orphanet Journal of Rare Diseases, 8 (1) p. 4. Peer-reviewed.
Brain damage in methylmalonic aciduria: 2-methylcitrate induces cerebral ammonium accumulation and apoptosis in 3D organotypic brain cell cultures.
Jafari P., Braissant O., Zavadakova P., Henry H., Bonafé L., Ballhausen D., 2013. Orphanet Journal of Rare Diseases, 8 (4) pp. 1-14. Peer-reviewed.
 
Different types of cell death are involved in brain damage of methylmalonic aciduria and glutaric aciduria type I
Zavadakova P., Jafari P., Cung HP, Werner D., Braissant O., Bonafé L., Ballhausen D., 2013. pp. S170-S171 dans ICIEM 2013, 12th International Congress of Inborn Errors of Metabolism, Journal of Inherited Metabolic Diseases.
Efficient Subtractive Cloning of Genes Activated by Lipopolysaccharide and Interferon γ in Primary-Cultured Cortical Cells of Newborn Mice.
Miyauchi O., Iwase K., Itoh K., Kato M., Seki N., Braissant O., Bachmann C., Shozu M., Sekiya S., Osada H. et al., 2013. Plos One, 8 (11) pp. e79236. Peer-reviewed.
 
Low concentrations of 3-hydroxy glutarate leads to ammonium accumulation and non-apoptotic cell death in developing brain cells
Jafari P., Zavadakova P., Cung H.P., Braissant O., Ballhausen D., 2013. pp. S173 dans ICIEM 2013, 12th International Congress of Inborn Errors of Metabolism, Journal of Inherited Metabolic Diseases.
 
BRAIN DAMAGE IN METHYLMALONIC ACIDURIA: 2-METHYLCITRATE LEADS TO AMMONIA INCREASE AND APOPTOSIS
Jafari P., Braissant O., Zavadakova P., Henry H., Bonafe L., Ballhausen D., 2012. pp. S9 dans Annual Symposium of the Society for the Study of Inborn Errors of Metabolism, Journal of Inherited Metabolic Diseases.
Creatine and guanidinoacetate transport at blood-brain and blood-cerebrospinal fluid barriers.
Braissant O., 2012. Journal of Inherited Metabolic Disease, 35 (4) pp. 655-664. Peer-reviewed.
 
Models of creatine deficiency syndromes by RNAi in developing brain cells
Braissant O., Hanna-El-Daher L., Beard E., Loup M., Henry H., 2012. p. 61 dans 16th Congress of the European-Federation-of-Neurological-Societies (EFNS), European Journal Of Neurology.
 
NEUROPATHOPHYSIOLOGICAL MECHANISMS IN GLUTARIC ACIDURIA TYPE I: 3-HYDROXYGLUTARIC ACID LEADS TO AMMONIA INCREASE AND NON-APOPTOTIC CELL DEATH
Jafari P., Braissant O., Zavadakova P., Henry H., Bonafe L., Ballhausen D., 2012. pp. S56 dans Annual Symposium of the Society for the Study of Inborn Errors of Metabolism, Journal of Inherited Metabolic Diseases.
 
CO-EXPRESSION OF GCDH AND OAT1 IN NEURONS AND PROXIMAL TUBULE CELLS
Ballhausen D., Jafari P., Bonafe L., Braissant O., 2011. pp. S141 dans Annual Symposium of the Society for the Study of Inborn Errors of Metabolism, Journal of Inherited Metabolic Diseases.
Creatine deficiency syndromes and the importance of creatine synthesis in the brain.
Braissant O., Henry H., Béard E., Uldry J., 2011. Amino Acids, 40 (5) pp. 1315-1324. Peer-reviewed.
Creatine, central nervous system and creatine deficiency syndromes
Braissant O., Uldry J., Béard E., 2011. pp. 189-196 dans Braissant O., Wakamatsu H., Kuo-Kang I., Allegaert K., Lenbury Y., Wachholtz A. (eds.) Recent Researches in Modern Medicine, WSEAS Press.
 
Methods to assess neuroinflammation.
Monnet-Tschudi F., Defaux A., Braissant O., Cagnon L., Zurich M.G., 2011. pp. Unit12.19 dans Current protocols in Toxicology chap. 12, Wiley.
 
P-glycoprotein modulation by valspodar and cyclosporin does not increase tumor uptake of doxorubicin administered via isolated lung perfusion to rats bearing sarcoma lung metastases.
Kuemmerle A., Yan H., Krueger T., Buclin T., Braissant O., Henry H., Ris H.B., Decosterd L.A., 2011. Anticancer Research, 31 (6) pp. 2121-2128.
 
The unsolved puzzle of neuropathogenesis in glutaric aciduria type I.
Jafari P., Braissant O., Bonafé L., Ballhausen D., 2011. Molecular Genetics and Metabolism, 104 (4) pp. 425-437. Peer-reviewed.
Ammonia toxicity to the brain: effects on creatine metabolism and transport and protective roles of creatine.
Braissant O., 2010. Molecular genetics and metabolism, 100 Suppl 1 pp. S53-8. Peer-reviewed.
 
Current concepts in the pathogenesis of urea cycle disorders.
Braissant Olivier, 2010. Molecular Genetics and Metabolism, 100 (Suppl 1) pp. S3-S12. Peer-reviewed.
 
DETERMINATION OF CELL-SPECIFIC NEUROTOXICITY OF MALONATE, METHYLMALONATE AND PROPIONATE IN A 3D RAT BRAIN CELL AGGREGATE SYSTEM
Ballhausen D., Henry H., Bonafe L., Braissant O., 2010. pp. S42 dans Annual Symposium of the Society for the Study of Inborn Errors of Metabolism, Journal of Inherited Metabolic Diseases.
 
DIFFERENTIAL EXPRESSION OF GLUTARYL-CoA DEHYDROGENASE IN ADULT RAT CNS, PERIPHERAL TISSUES AND DURING EMBRYONIC DEVELOPMENT
Ballhausen D., Bonafe L., Braissant O., 2010. pp. S41 dans Annual Symposium of the Society for the Study of Inborn Errors of Metabolism, Journal of Inherited Metabolic Diseases.
Dissociation of AGAT, GAMT and SLC6A8 in CNS: relevance to creatine deficiency syndromes.
Braissant O., Béard E., Torrent C., Henry H., 2010. Neurobiology of Disease, 37 (2) pp. 423-433. Peer-reviewed.
Synthesis and transport of creatine in the CNS: importance for cerebral functions.
Béard E., Braissant O., 2010. Journal of Neurochemistry, 115 pp. 297-313. Peer-reviewed.
CNTF protects oligodendrocytes from ammonia toxicity: intracellular signaling pathways involved.
Cagnon L., Braissant O., 2009. Neurobiology of Disease, 33 (1) pp. 133-142. Peer-reviewed.
Effects of the PPAR-beta agonist GW501516 in an in vitro model of brain inflammation and antibody-induced demyelination.
Defaux A., Zurich M.G., Braissant O., Honegger P., Monnet-Tschudi F., 2009. Journal of Neuroinflammation, 6 p. 15.
 
Evidence for catabolic pathway of propionate metabolism in CNS: expression pattern of methylmalonyl-CoA mutase and propionyl-CoA carboxylase alpha-subunit in developing and adult rat brain.
Ballhausen Diana, Mittaz Laureane, Boulat Olivier, Bonafé Louisa, Braissant Olivier, 2009. Neuroscience, 164 (2) pp. 578-587.
AGAT, GAMT and SLC6A8 distribution in the central nervous system, in relation to creatine deficiency syndromes: a review.
Braissant O., Henry H., 2008. Journal of Inherited Metabolic Disease, 31 (2) pp. 230-239. Peer-reviewed.
 
Ammonium alters creatine transport and synthesis in a 3D culture of developing brain cells, resulting in secondary cerebral creatine deficiency.
Braissant O., Cagnon L., Monnet-Tschudi F., Speer O., Wallimann T., Honegger P., Henry H., 2008. European Journal of Neuroscience, 27 (7) pp. 1673-1685.
 
Brain cells take up guanidinoacetate and convert it to creatine
Braissant O., Beard E., Torrent C., Henry H., 2008. pp. 69-69 dans Annual Symposium of the Society for the Study of Inborn Errors of Metabolism, Journal of Inherited Metabolic Disease. Peer-reviewed.
 
Knock-down of AGAT, GAMT and SLC6A8 by RNA interference in brain cells: New experimental models of creatine deficiencies in CNS
Beard E., Braissant O., 2008. pp. 69-69 dans Annual Symposium of the Society for the Study of Inborn Errors of Metabolism, Journal of Inherited Metabolic Disease. Peer-reviewed.
 
Methylmalonyl-CoA mutase expression in rat brain suggests autonomous methylamalonate production in CNS
Ballhausen D., Bonafe L., Boulat O., Braissant O., 2008. pp. S16 dans Annual Symposium of the Society for the Study of Inborn Errors of Metabolism, Journal of Inherited Metabolic Disease. Peer-reviewed.
Role of caspases, calpain and cdk5 in ammonia-induced cell death in developing brain cells.
Cagnon L., Braissant O., 2008. Neurobiology of Disease, 32 (2) pp. 281-292. Peer-reviewed.
 
Creatine and creatine kinase in health and disease - A bright future ahead?
Wyss M., Braissant O., Pischel I., Salomons G. S., Schulze A., Stöckler S., Wallimann T., 2007. Subcellular Biochemistry, 46 pp. 309-334.
 
Expression and function of AGAT, GAMT and CT1 in the mammalian brain
Braissant O., Bachmann C., Henry H., 2007. Subcellular Biochemistry, 46 pp. 67-81.
Hyperammonemia-induced toxicity for the developing central nervous system
Cagnon L., Braissant O., 2007. Brain Research Reviews, 56 (1) pp. 183-197.
 
Neurofilament proteins in brain diseases.
Braissant O., 2007. Brain Research Journal, 1 pp. 1-26.
 
Pathways depending on urea cycle intermediates
Braissant O., 2007. pp. 25-41 dans C Bachmann, J Häberle, H. G. Koch , J Leonard (eds.) Pathophysiology and Management of Hyperammonemia, SPS Publications.
 
Glial mechanisms of axonal growth protection from ammonia
Braissant O., Henry H., Cagnon L., Honegger P., Bachmann C., 2006. pp. 124-133 dans D. Haussinger , G. Kircheis , F. Schliess (eds.) Hepatic Encephalopathy and Nitrogen Metabolism, Springer.
Neurofilament proteins in brain diseases
Braissant O., 2006. pp. 25-51 dans R. K. Arlen (eds.) New Research on Neurofilament Proteins, Nova Science Publishers Inc..
 
Urea cycle disorders
Bachmann C., Braissant O., Cagnon L., Henry H., Boulat O., Kern I., 2006. pp. 313-319 dans D. Haussinger , G. Kircheis , F. Schliess (eds.) Hepatic Encephalopathy and Nitrogen Metabolism, Springer.
 
Cellular creatine transport: synopsis and reassessment
Speer O., Straumann N., Leuenberger T., Neukomm L. J., Braissant O., Henry H., Bachmann C., Wallimann T., 2005. pp. 64-86 dans C Jakobs, S Stöckler-Ipsiroglu, NM Verhoeven, GS Salomons (eds.) Clinical and Molecular Aspects of Defects in Creatine & Polyol Metabolism, SPS Verlagsgesellschaft.
Creatine synthesis and transport during rat embryogenesis: spatiotemporal expression of AGAT, GAMT and CT1.
Braissant O., Henry H., Villard A.M., Speer O., Wallimann T., Bachmann C., 2005. BMC Developmental Biology, 5 p. 9. Peer-reviewed.
 
Promoter rearrangements cause species-specific hepatic regulation of the glyoxylate reductase/hydroxypyruvate reductase gene by the peroxisome proliferator-activated receptor alpha.
Genolet R., Kersten S., Braissant O., Mandard S., Tan N.S., Bucher P., Desvergne B., Michalik L., Wahli W., 2005. Journal of Biological Chemistry, 280 (25) pp. 24143-24152. Peer-reviewed.
 
Synthesis and transport of creatine in the central nervous system
Braissant O., Villard A., Henry H., Speer O., Wallimann T., Bachmann C., 2005. pp. 49-63 dans C Jakobs, S Stöckler-Ipsiroglu, NM Verhoeven, GS Salomons (eds.) Clinical and Molecular Aspects of Defects in Creatine & Polyol Metabolism, SPS Verlagsgesellschaft.
 
Unusual astrocyte reactivity caused by the food mycotoxin ochratoxin A in aggregating rat brain cell cultures.
Zurich M.G., Lengacher S., Braissant O., Monnet-Tschudi F., Pellerin L., Honegger P., 2005. Neuroscience, 134 (3) pp. 771-782.
Ammonia toxicity to the brain and creatine.
Bachmann C., Braissant O., Villard A.M., Boulat O., Henry H., 2004. Molecular Genetics and Metabolism, 81 (Suppl 1) pp. S52-S57. Peer-reviewed.
 
Measurement of nitric oxide-related enzymes in the brain by in situ hybridization
Braissant O., 2004. Methods in Molecular Biology, 279 pp. 113-124.
Tumor necrosis factor-alpha and alphaB-crystallin up-regulation during antibody-mediated demyelination in vitro: a putative protective mechanism in oligodendrocytes.
Duvanel C.B., Monnet-Tschudi F., Braissant O., Matthieu J.M., Honegger P., 2004. Journal of Neuroscience Research, 78 (5) pp. 711-722.
 
From arginine to creatine: Regional and cellular gene expression of enzymes and transporters linking nitrogen and energy metabolism in brain
Bachmann C., Henry H., Braissant O., 2003. pp. 153-165 dans Bachmann C., Koletzko B. (eds.) Genetic Expression and Nutrition, Lippincott Williams & Wilkins.
Ammonium-induced impairment of axonal growth is prevented through glial creatine.
Braissant O., Henry H., Villard A.M., Zurich M.G., Loup M., Eilers B., Parlascino G., Matter E., Boulat O., Honegger P. et al., 2002/11/15. The Journal of neuroscience, 22 (22) pp. 9810-9820. Peer-reviewed.
Alteration of amino acid metabolism in neuronal aggregate cultures exposed to hypoglycaemic conditions.
Honegger P., Braissant O., Henry H., Boulat O., Bachmann C., Zurich M.G., Pardo B., 2002/06. Journal of Neurochemistry, 81 (6) pp. 1141-1151.
 
Expression and localization of PPARs in the rat ovary during follicular development and the periovulatory period.
Komar C.M., Braissant O., Wahli W., Curry T.E., 2001/11. Endocrinology, 142 (11) pp. 4831-4838.
 
Rat PPARs: quantitative analysis in adult rat tissues and regulation in fasting and refeeding.
Escher P., Braissant O., Basu-Modak S., Michalik L., Wahli W., Desvergne B., 2001/10. Endocrinology, 142 (10) pp. 4195-4202.
 
Connexins 40 and 43 are differentially regulated within the kidneys of rats with renovascular hypertension.
Haefliger J.A., Demotz S., Braissant O., Suter E., Waeber B., Nicod P., Meda P., 2001/07. Kidney International, 60 (1) pp. 190-201. Peer-reviewed.
 
Differential involvement of peroxisome-proliferator-activated receptors alpha and delta in fibrate and fatty-acid-mediated inductions of the gene encoding liver fatty-acid-binding protein in the liver and the small intestine.
Poirier H., Niot I., Monnot M.C., Braissant O., Meunier-Durmort C., Costet P., Pineau T., Wahli W., Willson T.M., Besnard P., 2001/04. Biochemical Journal, 355 (Pt 2) pp. 481-488.
Differential expression of the cationic amino acid transporter 2(B) in the adult rat brain
Braissant O., Gotoh T., Loup M., Mori M., Bachmann C., 2001. Brain Research. Molecular Brain Research, 91 (1-2) pp. 189-195.
Endogenous synthesis and transport of creatine in the rat brain: an in situ hybridization study
Braissant O., Henry H., Loup M., Eilers B., Bachmann C., 2001. Brain Research. Molecular Brain Research, 86 (1-2) pp. 193-201.
 
Cellular localization, expression and regulation of neuropeptide Y in kidneys of hypertensive rats.
Haefliger J.A., Waeber B., Grouzmann E., Braissant O., Nussberger J., Nicod P., Waeber G., 1999. Regulatory peptides, 82 (1-3) pp. 35-43. Peer-reviewed.
Hyperammonemia: regulation of argininosuccinate synthetase and argininosuccinate lyase genes in aggregating cell cultures of fetal rat brain.
Braissant O., Honegger P., Loup M., Iwase K., Takiguchi M., Bachmann C., 1999. Neuroscience Letters, 266 (2) pp. 89-92.
L-arginine uptake, the citrulline-NO cycle and arginase II in the rat brain: an in situ hybridization study
Braissant O., Gotoh T., Loup M., Mori M., Bachmann C., 1999. Brain Research. Molecular Brain Research, 70 (2) pp. 231-241.
Peroxisome proliferator-activated receptor beta regulates acyl-CoA synthetase 2 in reaggregated rat brain cell cultures.
Basu-Modak S., Braissant O., Escher P., Desvergne B., Honegger P., Wahli W., 1999. Journal of Biological Chemistry, 274 (50) pp. 35881-35888.
 
Differential expression of peroxisome proliferator-activated receptor-alpha, -beta, and -gamma during rat embryonic development.
Braissant O., Wahli W., 1998/06. Endocrinology, 139 (6) pp. 2748-2754.
A simplified in situ hybridization protocol using non-radioactively labelled probes to detect abundant and rare mRNAs on tissue sections
Braissant O., Wahli W., 1998. Biochemica, 1 pp. 10-16.
 
Fatty acids, eicosanoids, and hypolipidemic agents identified as ligands of peroxisome proliferator-activated receptors by coactivator-dependent receptor ligand assay.
Krey G., Braissant O., L'Horset F., Kalkhoven E., Perroud M., Parker M.G., Wahli W., 1997/06. Molecular Endocrinology, 11 (6) pp. 779-791.
 
9-cis-retinoic acid enhances fatty acid-induced expression of the liver fatty acid-binding protein gene.
Poirier H., Braissant O., Niot I., Wahli W., Besnard P., 1997. Febs Letters, 412 (3) pp. 480-484.
 
Retinoid X receptor and peroxisome proliferator-activated receptor activate an estrogen responsive gene independent of the estrogen receptor.
Nuñez S.B., Medin J.A., Braissant O., Kemp L., Wahli W., Ozato K., Segars J.H., 1997. Molecular and Cellular Endocrinology, 127 (1) pp. 27-40.
 
PPAR tissue distribution and interactions with other hormone-signaling pathways.
Lemberger T., Braissant O., Juge-Aubry C., Keller H., Saladin R., Staels B., Auwerx J., Burger A.G., Meier C.A., Wahli W., 1996/12. Annals of the New York Academy of Sciences, 804 pp. 231-251.
 
Differential expression of peroxisome proliferator-activated receptors (PPARs): tissue distribution of PPAR-alpha, -beta, and -gamma in the adult rat.
Braissant O., Foufelle F., Scotto C., Dauça M., Wahli W., 1996/01. Endocrinology, 137 (1) pp. 354-366.
 
Peroxisome proliferator activated receptors: transcriptional regulators of adipogenesis, lipid metabolism and more....
Wahli W., Braissant O., Desvergne B., 1995/05. Chemistry and Biology, 2 (5) pp. 261-266.
 
Ribosomal protein L27 is identical in chick and rat.
Lebeau M.C., Alvarez-Bolado G., Braissant O., Wahli W., Catsicas S., 1991/03. Nucleic Acids Research, 19 (6) p. 1337.
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